1. Digitum: Repositorio Institucional de la Universidad de Murcia
  2. Revistas y Congresos
  3. Revistas
  4. Histology and histopathology
  5. Vol.28, nº 6 (2013)
Por favor, use este identificador para citar o enlazar este ítem: http://hdl.handle.net/10201/58401

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dc.contributor.authorShin, Taekyun-
dc.contributor.authorAhn, Meejung-
dc.contributor.authorMatsumoto, Yoh-
dc.contributor.authorMoon, Changjong-
dc.date.accessioned2018-05-15T11:28:29Z-
dc.date.available2018-05-15T11:28:29Z-
dc.date.issued2013-
dc.identifier.citationHistology and Histopathology, vol. 28, nº 6, (2013)es
dc.identifier.issn1699-5848-
dc.identifier.issn0213-3911-
dc.identifier.urihttp://hdl.handle.net/10201/58401-
dc.description.abstractHuman peripheral demyelinating diseases, such as Guillain-Barré syndrome (GBS), are characterized by inflammation and demyelination in the peripheral nervous system. Similarities in the pathology between GBS and the animal model of experimental autoimmune neuritis (EAN) indicate that autoimmune responses are involved in both diseases. This article summarizes the general aspects of the EAN model in Lewis rats and discusses the potential role of macrophages in the progression of EAN. A better understanding of macrophages may help to design alternative therapeutic strategies for organ-specific autoimmune diseases, including GBS.es
dc.formatapplication/pdfes
dc.format.extent6es
dc.languageenges
dc.publisherF. Hernández y Juan F. Madrid. Universidad de Murcia: Departamento de Biología Celular e Histologíaes
dc.rightsinfo:eu-repo/semantics/openAccesses
dc.subjectAlternatively activated macrophageses
dc.subjectExperimental autoimmune neuritises
dc.subjectGuillain-Barré syndromees
dc.subjectPeripheral nervous system inflammationes
dc.subject.otherCDU::6 - Ciencias aplicadas::61 - Medicinaes
dc.titleMechanism of experimental autoimmune neuritis in Lewis rats: the dual role of macrophageses
dc.typeinfo:eu-repo/semantics/articlees
Aparece en las colecciones:Vol.28, nº 6 (2013)

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