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dc.contributor.authorTaranto, E.es
dc.contributor.authorLeech, M.es
dc.date.accessioned2011-06-30T12:02:16Z-
dc.date.available2011-06-30T12:02:16Z-
dc.date.issued2006-
dc.identifier.issn0213-3911es
dc.identifier.urihttp://hdl.handle.net/10201/22621-
dc.description.abstractRheumatoid Arthritis (RA) is a chronic disease characterised by synovial lining hyperplasia and progressive destruction of joint tissues. Experimental data suggests that abnormal alterations in the expression of proteins involved in maintaining homeostatic control of the cell cycle is involved in disease progression in RA. By contributing to the overgrowth of synovial tissue, factors such as dysregulated proliferation or reduced apoptosis of cells can directly influence the pathological outcome of RA.es
dc.formatapplication/pdfes
dc.format.extent7es
dc.languageenges
dc.publisherMurcia : F. Hernándezes
dc.relation.ispartofHistology and histopathologyes
dc.rightsinfo:eu-repo/semantics/openAccesses
dc.subjectCell cyclees
dc.subjectApoptosises
dc.subject.otherCDU::6 - Ciencias aplicadas::61 - Medicinaes
dc.titleExpression and function of cell cycle proteins in rheumatoid arthritis synovial tissuees
dc.typeinfo:eu-repo/semantics/articlees
Aparece en las colecciones:Vol.21, nº 2 (2006)

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