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dc.contributor.authorKrutovskikh, V.es
dc.contributor.authorYamasaki, H.es
dc.date.accessioned2011-02-22T10:41:40Z-
dc.date.available2011-02-22T10:41:40Z-
dc.date.issued1997-
dc.identifier.issn0213-3911es
dc.identifier.urihttp://hdl.handle.net/10201/18971-
dc.description.abstractThere is a growing body of evidence supporting the etiologic implication of gap junctional intercellular communication disorders in carcinogenesis. Substantial progress has recently be.en made both in molecular biology of gap junction and in the field of cancer research. They provide new insights and conceptions of gap junctional disorders in tumor pathology. Modern understanding of the structure, function and regulation of gap junctions, as well as putative mechanisms of its disorders in human and experimental carcinogenesis are discussed in this review with particular emphasis on fast-moving aspects of this problem.es
dc.formatapplication/pdfes
dc.format.extent8es
dc.languageenges
dc.publisherMurcia : F. Hernándezes
dc.relation.ispartofHistology and histopathologyes
dc.rightsinfo:eu-repo/semantics/openAccesses
dc.subjectGap junctiones
dc.subjectConnexines
dc.subject.otherCDU::6 - Ciencias aplicadas::61 - Medicina::616 - Patología. Medicina clínica. Oncologíaes
dc.titleThe role of gap junctional intercellular communication (GJIC) disorders in experimental and human carcinogenesises
dc.typeinfo:eu-repo/semantics/articlees
Aparece en las colecciones:Vol.12, nº 3 (1997)



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