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dc.contributor.authorZhu, Mingzhu-
dc.contributor.authorDu, Junbao-
dc.contributor.authorLiu, Angie Dong-
dc.contributor.authorHolmberg, Lukas-
dc.contributor.authorTang, Chaoshu-
dc.contributor.authorJin, Hongfang-
dc.date.accessioned2019-09-25T14:43:28Z-
dc.date.available2019-09-25T14:43:28Z-
dc.date.issued2014-
dc.identifier.citationHistology and Histopathology, vol. 29, nº 9, (2014)es
dc.identifier.issn1699-5848-
dc.identifier.issn0213-3911-
dc.identifier.urihttp://hdl.handle.net/10201/74886-
dc.description.abstractIn the middle of the 1980s, nitric oxide received extensive attention because of its significant effects in life science. Then, carbon monoxide and hydrogen sulfide were discovered to be gasotransmitters playing important roles in regulating cellular homeostasis. As a common air pollutant, sulfur dioxide (SO2) can cause great harm to the human body by producing free radicals, which causes oxidative damage to various organs. Recently, endogenous SO2 was found to be produced in the cardiovascular system and might be a bioactive molecule regulating the physiological activities including cardiovascular oxidative stress.es
dc.formatapplication/pdfes
dc.format.extent5es
dc.languageenges
dc.publisherF. Hernández y Juan F. Madrid. Universidad de Murcia: Departamento de Biología Celular e Histologíaes
dc.rightsinfo:eu-repo/semantics/openAccesses
dc.rightsAttribution-NonCommercial-NoDerivatives 4.0 International*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/4.0/*
dc.subjectSulfur dioxidees
dc.subjectCardiovasculares
dc.subjectOxidative stresses
dc.subject.otherCDU::5 - Ciencias puras y naturales::57 - Biología::576 - Biología celular y subcelular. Citologíaes
dc.titleEffect of endogenous sulfur dioxide in regulating cardiovascular oxidative stresses
dc.typeinfo:eu-repo/semantics/articlees
Aparece en las colecciones:Vol.29, nº 9 (2014)

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