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Campo DC | Valor | Lengua/Idioma |
---|---|---|
dc.contributor.author | Di Rosa, G. | - |
dc.contributor.author | Puzzo, D. | - |
dc.contributor.author | Sant'Angelo, A. | - |
dc.contributor.author | Trinchese, F. | - |
dc.contributor.author | Arancio, O. | - |
dc.date.accessioned | 2016-12-21T15:21:23Z | - |
dc.date.available | 2016-12-21T15:21:23Z | - |
dc.date.created | 2003 | - |
dc.date.issued | 2003 | - |
dc.identifier.uri | http://hdl.handle.net/10201/51603 | - |
dc.description.abstract | Alpha-synuclein belongs to a family of vertebrate proteins, encoded by three different genes: a, ß, and g. The protein has become of interest to the neuroscience community in the last few years after the discovery that a mutation in the a-synuclein gene is associated with familial autosomal-dominant early-onset forms of Parkinson Disease. However, it is not yet clear how the protein is involved in the disease. Several studies have suggested that a-synuclein plays a role in neurotransmitter release and synaptic plasticity. This hypothesis might help elucidate how a-synuclein malfunctioning contributes to the development of a series of disorders known as synucleinopathies. | es |
dc.format | application/pdf | es |
dc.format.extent | 9 | es |
dc.language | eng | es |
dc.relation.ispartofseries | Vol. 18 | es |
dc.relation.ispartofseries | Nº. 4 | es |
dc.rights | info:eu-repo/semantics/openAccess | es |
dc.rights | An error occurred on the license name. | * |
dc.rights.uri | An error occurred getting the license - uri. | * |
dc.subject | Synuclein | es |
dc.subject | Neurotransmission | es |
dc.subject.other | CDU::5 - Ciencias puras y naturales::57 - Biología | es |
dc.title | a-synuclein: between synaptic function and dysfunction | es |
dc.type | info:eu-repo/semantics/article | es |
Aparece en las colecciones: | Vol.18, nº 4 (2003) |
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