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dc.contributor.authorGreenberg, Stephen R.es
dc.date.accessioned2011-02-01T08:45:06Z-
dc.date.available2011-02-01T08:45:06Z-
dc.date.issued1990-
dc.identifier.issn0213-3911es
dc.identifier.urihttp://hdl.handle.net/10201/18192-
dc.description.abstractAn experimental murine model of chronic lead retention is established. This model excludes joint communication and synovial fluid solubility of lead ions recognized in human gunshot injury. Degenerative changes in the visceral organs and bone appear in these mice 30 days after implantation of lead spheres in the subcutaneous tissues of the posterior body wall. Degeneration, necrosis and interstitial fibrosis of the lung, kidney and remodeling of femoral bone are noted during the course of the experiment (120 days). It is concluded that the systemic diffusion of lead ions from a central focus is related to the development of an absorptive circulatory mechanism around the retained peripheral lead mass.es
dc.formatapplication/pdfes
dc.format.extent6es
dc.languageenges
dc.publisherMurcia : F. Hernándezes
dc.relation.ispartofHistology and histopathologyes
dc.rightsinfo:eu-repo/semantics/openAccesses
dc.subjectLead intoxicationes
dc.subjectRetained bulletes
dc.subject.otherCDU::6 - Ciencias aplicadas::61 - Medicina::616 - Patología. Medicina clínica. Oncologíaes
dc.titleThe histopathology of tissue lead retentiones
dc.typeinfo:eu-repo/semantics/articlees
Aparece en las colecciones:Vol. 5, nº 4 (1990)

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